南方医科大学学报 ›› 2026, Vol. 46 ›› Issue (9): 2023-2034.doi: 10.12122/j.issn.1673-4254.2026.09.03
• • 上一篇
叶丽旋1(
), 廖雨欣2, 刘琼2, 张佳2, 高磊2, 董钊扬1(
)
收稿日期:2026-01-20
出版日期:2026-09-20
发布日期:2026-09-30
通讯作者:
董钊扬
E-mail:2669556676@qq.com;021092@ gzucm.edu.cn
作者简介:叶丽旋,在读硕士研究生,E-mail: 2669556676@qq.com
基金资助:
Lixuan YE1(
), Yuxin LIAO2, Qiong LIU2, Jia ZHANG2, Lei GAO2, Zhaoyang DONG1(
)
Received:2026-01-20
Online:2026-09-20
Published:2026-09-30
Contact:
Zhaoyang DONG
E-mail:2669556676@qq.com;021092@ gzucm.edu.cn
Supported by:摘要:
目的 探讨逍遥散(XYS)在秀丽隐杆线虫(C. elegans)帕金森病(PD)模型中的神经保护作用及其分子机制。 方法 采用6-羟基多巴胺(6-OHDA)诱导建立PD线虫模型,并结合α-突触核蛋白(α-syn)过表达转基因虫株(NL5901)进行验证。将线虫分为对照组、模型组及XYS低(25 μg/mL)、中(50 μg/mL)、高(100 μg/mL)剂量组。通过生存曲线分析评估寿命;采用20 s内身体弯曲次数及基础减慢反应分别评价线虫的运动功能和食物相关行为;利用GFP标记的转基因虫株观察多巴胺能神经元损伤及α-syn聚集水平;采用二氢乙啶荧光探针(DHE)检测体内活性氧(ROS)水平,并结合RT-qPCR分析dat-1、cat-2、ced-9、gst-4及sod-3等相关基因表达;通过RNA干扰(RNAi)沉默skn-1表达,以验证XYS作用是否依赖于SKN-1/Nrf2信号通路。 结果 XYS可显著延长PD模型线虫寿命(P<0.001),增加第5天和第10天的20 s内身体弯曲次数(P<0.05),并改善基础减慢反应(P<0.05);XYS可减轻6-OHDA诱导的多巴胺能神经元损伤,提高dat-1、cat-2及ced-9表达水平(P<0.05),并降低α-syn聚集水平(P<0.001)。此外,XYS处理后降低ROS水平(P<0.05),sod-3及gst-4等抗氧化相关基因表达上调(P<0.05)。机制研究显示,XYS可增强skn-1::GFP及sod-3::GFP荧光信号(P<0.05)。在skn-1 RNAi条件下,脂质含量下降(P<0.05),且XYS对上述改变无明显改善作用(P>0.05);同时,XYS对sod-3::GFP荧光降低的恢复作用消失(P>0.05)。 结论 XYS可减轻氧化应激并抑制α-syn异常聚集,从而改善秀丽隐杆线虫PD模型神经功能,其作用可能与SKN-1/Nrf2相关抗氧化通路有关。
叶丽旋, 廖雨欣, 刘琼, 张佳, 高磊, 董钊扬. 逍遥散通过SKN-1抗氧化通路改善秀丽隐杆线虫帕金森病模型α-突触核蛋白聚集和神经元损伤[J]. 南方医科大学学报, 2026, 46(9): 2023-2034.
Lixuan YE, Yuxin LIAO, Qiong LIU, Jia ZHANG, Lei GAO, Zhaoyang DONG. Xiaoyao San alleviates abnormal α‑syn aggregation and neuronal injury in a Caenorhabditis elegans model of Parkinson disease via the SKN-1 antioxidative pathway[J]. Journal of Southern Medical University, 2026, 46(9): 2023-2034.
| Time (min) | Flow (mL/min) | % B |
|---|---|---|
| 0.00 | 0.30 | 5.0 |
| 2.00 | 0.30 | 5.0 |
| 18.00 | 0.30 | 95.0 |
| 20.00 | 0.30 | 95.0 |
表1 色谱梯度程序
Tab.1 Chromatography gradient program
| Time (min) | Flow (mL/min) | % B |
|---|---|---|
| 0.00 | 0.30 | 5.0 |
| 2.00 | 0.30 | 5.0 |
| 18.00 | 0.30 | 95.0 |
| 20.00 | 0.30 | 95.0 |
| Gene | Forward primer (5'-3') | Reverse primer (5'-3') |
|---|---|---|
| Dat-1 | TTGGTGCCTACAGACGATCC | GCTGTTCCGTCTTCTGACCA |
| Cat-2 | ACTGAAGAACCTGCGGTACG | CCGATGGTGATTGCTCCTGC |
| Gst-4 | GATGCTCGTGCTCTTGCTGA | GGAGTCGTTGGCTTCAGCTT |
| Ced-9 | TGCTCAGGACTTGCCATCAC | GCGTGAAATAGTCGACCACA |
| Sod-3 | ATCTACTGCTCGCACTGCTT | ACGTAGGTGGCATGATGCTT |
| β-actin | ACGACGAGTCCGGCCCATCC | GAAAGCTGGTGGTGACGATGGTT |
表2 目的基因引物序列
Tab.2 Primer sequences of target genes
| Gene | Forward primer (5'-3') | Reverse primer (5'-3') |
|---|---|---|
| Dat-1 | TTGGTGCCTACAGACGATCC | GCTGTTCCGTCTTCTGACCA |
| Cat-2 | ACTGAAGAACCTGCGGTACG | CCGATGGTGATTGCTCCTGC |
| Gst-4 | GATGCTCGTGCTCTTGCTGA | GGAGTCGTTGGCTTCAGCTT |
| Ced-9 | TGCTCAGGACTTGCCATCAC | GCGTGAAATAGTCGACCACA |
| Sod-3 | ATCTACTGCTCGCACTGCTT | ACGTAGGTGGCATGATGCTT |
| β-actin | ACGACGAGTCCGGCCCATCC | GAAAGCTGGTGGTGACGATGGTT |
图2 XYS干预能够延长PD线虫寿命并改善其活动能力
Fig.2 XYS extends lifespan and improves locomotor activity of C. elegans PD models. A: Survival curves of C. elegans exposed to different concentrations of XYS. B: Schematic diagram of the experimental design for combined 6-OHDA and XYS treatment. C: Survival curves of 6-OHDA-induced PD model worms with or without XYS treatment. D: Quantification of the basal slowing response. E: Quantification of body bends within 20 s on days 5 and 10 post-treatment. Data represent Mean±SD from at least 3 independent biological replicates (n=50 for lifespan assays and n=30 for other assays). #P<0.05, ##P<0.01 vs control group; *P<0.05, **P<0.01, ***P<0.001, ****P<0.0001 vs model group.
图3 XYS干预能够延长PD转基因模型NL5901线虫寿命并改善其体内的α-syn聚集
Fig.3 XYS extends lifespan and reduces α-syn aggregation in NL5901 worms. A: Survival curves of NL5901 worms with or without XYS treatment. B: Quantification of body bends within 20 s on days 5 and 10 post-treatment. C: Representative images of α‑syn::YFP fluorescence in body wall muscle cells and quantification of fluorescence intensity (Scale bar=100 μm). Data represent Mean±SD from at least 3 independent biological replicates (n=50 for lifespan assays and n=30 for other assays). *P<0.05, **P<0.01, ***P<0.001, ****P<0.0001 vs control group.
图4 XYS干预显著改善6-OHDA诱导的PD线虫模型多巴胺能神经元变性
Fig.4 XYS protects against 6-OHDA-induced dopaminergic neurodegeneration in C. elegans. A: Representative GFP images of dopaminergic neurons with or without XYS treatment and quantification of fluorescence intensity (Scale bar=100 μm). B: Relative mRNA expression levels of dat-1, cat-2, and ced-9. Data represent Mean±SD from at least three independent biological replicates (n=30 for fluorescence analysis and n=3 for qPCR analysis). ##P<0.01, ###P<0.001, ####P<0.0001 vs control group; **P<0.01, ***P<0.001, ****P<0.0001 vs model group.
图5 XYS干预能够显著改善线虫体内抗氧化效应
Fig.5 XYS significantly enhances antioxidant capacity in C. elegans. A: Representative fluorescence images of sod-3::GFP worms and quantification of sod-3::GFP fluorescence intensity (Scale bar=100 μm). B: Relative mRNA expression levels of sod-3 and gst-4. Data represent Mean±SD from at least 3 independent biological replicates (n=30 for fluorescence analysis and n=3 for qPCR analysis). *P<0.05, **P<0.01, ***P<0.001, ****P<0.0001 vs control group.
图6 XYS干预能够显著降低不同PD线虫模型体内氧化应激水平
Fig.6 XYS significantly reduces oxidative stress levels in different C. elegans models of PD. A: Representative DHE staining images of NL5901 worms with or without XYS treatment and quantification of DHE fluorescence intensity. B: Representative DHE staining images of 6-OHDA-treated N2 worms with or without XYS treatment and quantification of DHE fluorescence intensity. Scale bar=100 μm. Data represent Mean±SD from at least 3 independent biological replicates (n=30). ####P<0.0001 vs control group; *P<0.05, **P<0.01 vs model group.
图7 XYS通过上调SKN-1改善PD线虫模型抗氧化效应
Fig.7 XYS improves antioxidant responses in PD C. elegans models through upregulation of SKN-1. A: Representative fluorescence images of LD1 worms. B: Quantification of fluorescence intensity in LD1 worms. C: Quantification of fluorescence intensity in CF1553 worms. D: Representative fluorescence images of CF1553 worms. E: Representative fluorescence images of skn-1 RNAi-treated worms and quantification of fluorescence intensity. Scale bar=100 μm. Data represent Mean±SD from at least 3 independent biological replicates (n=30). ##P<0.01, ###P<0.001 vs control group; #P<0.05 vs skn-1 RNAi group; *P<0.05, **P<0.01 vs model group.
图8 XYS干预能够改善PD线虫模型脂质代谢异常
Fig.8 XYS ameliorates lipid metabolic dysfunction in PD C. elegans models. A: Representative fluorescence images of LIU104 worms and quantification of fluorescence intensity. B: Representative fluorescence images of DMS303 worms and quantification of fluorescence intensity. C: Representative Nile Red staining images of NL5901 worms and quantification of fluorescence intensity. Scale bar=100 μm. Data represent Mean±SD from at least 3 independent biological replicates (n=30). #P<0.05, ##P<0.01 vs control group; *P<0.05, **P<0.01 vs model group.
图9 XYS通过skn-1调节α-syn蓄积及脂质积累
Fig.9 XYS regulates α-synuclein accumulation and lipid deposition via skn-1. A: Representative fluorescence images of skn-1 RNAi worms and quantification of fluorescence intensity. B: Representative Nile Red staining images of skn-1 RNAi worms and quantification of lipid accumulation based on Nile Red staining. Scale bar=100 μm. Data represent Mean±SD from at least 3 independent biological replicates (n=30). ##P<0.01, ###P<0.001 vs control group.
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