[1] Loeffler J, Hagmeyer L, Hebart H, et al. Rapid detection of point mutations by fluorescence resonance energy transfer and probe melting curves in Candida species[J]. Clin Chem, 2000, 46(5):631-5. [2] White TC, Holleman S, Dy F, et al. Resistance mechanisms in clinical isolates of Candida albicans[J]. Antimicrob Agents Chemother,2002, 46(6): 1704-13. [3] 王应斌,王宏,郭辉玉,等.应用M13引物PCR指纹法分析假丝酵母菌氟康唑耐药性[J].第一军医大学学报,2003,23(12):1303-6.Wang YB, Wang H, Guo HY, et al. Fluconazole resistance in Candida albicans assayed by PCR fingerprinting with M13 prime.[J]. J First Mil Med Univ/Di Yi Jun Yi Da Xue Xue Bao, 2003, 23(12): 1303-6. [4] Peters S, Koschinsky S, Schwieger F, et al. Succession of microbial communities during hot composting as detected by PCR-singlestrand-conformation polymorphism-based genetic profiles of small-subunit rRNA genes[J]. Appl Environ Microbiol, 2000, 66(3): 930-6. [5] Perea S, Lopez-Ribot JL, Kirkpatrick WR, et al. Prevalence of molecular mechanisms of resistance to azole antifungal agents in Candida albicans strains displaying high-level fluconazole resistance isolated from human immunodeficiency virus-infected patients[J].Antimicrob Agents Chemother, 2001, 45(10): 2676-84. [6] AoyamaY, KudoM, AsaiK, et al. Emergence of fluconazoleresistant sterol 14-demethylase P450 (CYPS1) in Candida albicans is a model demonstrating the diversification mechanism of P450[J].Arch Biochem Biophys, 2000, 379(1): 170-1. [7] Walsh TJ, Francesconi A, Kasai M, et al. PCR and single-strand conformational polymorphism for recognition of medically important opportunistic fungi[J]. J Clin Microbiol, 1995, 33(12): 3216-20. [8] Sanglard D, Ischer F, Koymans L, et al. Amino acid substitutions in the cytochrome P-450 lanosterol 14alpha-demethylase (CYP51A 1)from azole-resistant Candida albicans clinical isolates contribute to resistance to azole antifungal agents[J]. Antimicrob Agents Chemother,1998, 42(2): 241-53. [9] 龙丰,张永信,兰和魁,等.耐氟康唑白色念珠菌ERG11的基因突变[J].中华传染病杂志,2002,20(4):211-4.Long F, Zhang YX, Lan HK, etal. The point mutation of cytochrome P-450 lanosterol 14-α demethylase ERG11 gene in fluconazole resistant Candida albicans[J]. Chin J Infect Dis, 2002,20(4): 211-4. [10] Song JL, Harry JB, Eastman RT, et al. The Candida albicans lanosterol 14-alpha-demethylase (ERG11) gene promoter is maximally induced after prolonged growth with antifungal drugs[J].Antimicrob Agents Chemother, 2004, 48(4): 1136-44. [11] Marr KA, Lyons CN, Rustad TR, et al. Rapid, transient fluconazole resistance in Candida albicans is associated with increased mRNA levels of CDR[J]. Antimicrob Agents Chemother, 1998, 42(10):2584-9. [12] Barker KS, Crisp S, Wiederhold N, et al. Genome-wide expression profiling reveals genes associated with amphotericin B and fluconazole resistance in experimentally induced antifungal resistant isolates of Candida albicans[J]. J Antimicrob Chemother, 2004, 54(2): 376-85. [13] Karababa M, Coste AT, Rognon B, et al. Comparison of gene expression profiles of Candida albicans azole-resistant clinical isolates and laboratory strains exposed to drugs inducing multidrug transporters[J]. Antimicrob Agents Chemother, 2004, 48(8): 3064-79. |