[1] 屈丽君.小儿急性光气中毒42例临床分析[J].安徽医学,2003,24(4):21-3Qu LJ.Clinical analysis of 42 children poisoning phosgene[J].An Hui Med,2003,24(4):21-3. [2] 王招兄,李思惠.急性光气中毒112例临床分析[J].化工劳动保护(工业卫生与职业病分册),1997,18(5):232. [3] 杨焱,君彦明,杨文润,等.33例急性光气中毒抢救成功的体会[J].中国煤炭工业医学杂志,1998,1(1):39.Yang Y,Jun YM,Yang WR,et al.Successful rescue of 33 cases poisoning phosgene. Chin J Coal Industry Med, 1998, 1(1): 39. [4] Sciuto AM. Disruption of gas exchange in mice after exposure to the chemical threat agent phosgene[J]. Mil Med, 2001, 166(9): 809-21. [5] 隰建成,周宝桐.脓毒症过程中的炎症介质与细胞凋亡关系的研究进展[J].中国危重病急救医学,2000,12(6):378-80.Cheng JC, Zhou BT. Progress of relation between inflammation medium and apoptosis in sepsis proceeding[J]. Chin Criti Care Med,2000, 12(6): 378-80. [6] Tesfaigzi J, Wood MB, Johnson NF, et al. Apoptosis is a pathway responsible for the resolution of endotoxin induced alveolar type Ⅱcell hyperplasia in the rat[J]. Love Respir Res Ins, 1998, 79(5):303-11. [7] Gruener S, Dieter C, Walter E. Culture and transformation of human airway epithelial cells[J]. Am J Physiol, 1995, 268 (Lung Cell Mol Physiol 12): L347-60. [8] 张玉琴,欧阳静萍,李端详,等.硫氮卓酮对失血性休克犬肺泡Ⅱ型细胞的影响[J].中国现代医学杂志,2000,10(6):9-11.Zhang YQ, Ou YJP, Li DX, et al. Influence of diltiazem to the alveolus cell of the hemorrhagic shock dogs[J]. Chin J Modem Med, 2000, 10(6): 9-11. [9] Abu-Bakar S, Shafee N, Chee HY. Induction ofapoptosis in neurogenie pulmonary edema[J]. Med J Malaysia, 1999, 54(3): 402-3. [10] 李文丽,海春旭,张晓迪,等.光气诱导小鼠原代肺Ⅱ型细胞凋亡[J].第四军医大学学报,2004,25(3):229-32.Li WL, Hai CX, Zhang XD, et al. Apoptosis of alveolar type Ⅱ cell induced by phosgene in mice[J]. J Fourth Mil Med Univ, 2004, 25(3): 229-32. |