[1] Shah PB, Lilly CM. Interventional therapy for coronary artery disease[J]. Am J Respir Crit Care Med, 2002, 166(6): 791-6. [2] Peters S, Gotting B, Trummel M, et al. Valsartan for prevention of restenosis after stenting of type B2/C lesions: the VAL-PREST trial[J]. J Invasive Cardiol, 2001, 13(2): 93-7. [3] Zuckerbraun BS, Tzeng E. Vascular Gene Therapy: A reality of the 21st century[J]. Arch Surg, 2002, 137(7): 854-61 [4] Laukkanen MO, Kivela A, Rissanen T, et al. Adenovims-mediated extracellular superoxide dismutase gene therapy reduces neointima formation in balloon-denuded rabbit aorta[J]. Circulation, 2002, 106(15):1999-2003. [5] Yla-Herttuala S, Martin JF. Cardiovascular gene therapy[J]. Lancet,2000, 355(9199): 213-22. [6] Zhang Z, Xiao Z, Diamond SL. Shear stress induction of C-type natriuretic peptide (CNP) in endothelial cells is independent of NO autocrine signaling[J]. Ann Biomed Eng, 1999, 27(4): 419-26. [7] Casco VH, Veinot JP, Kuroski de Bold ML, et al. Natriuretic peptide system gene expression in human coronary arteries[J]. J Histochem Cytochem, 2002, 50(6): 799-809. [8] 欧阳平,刘伊丽,许顶立,等.p53、Rb、IGF-I、AIB基因转移对血管新生内膜增殖的影响[J].第一军医大学学报,2001,21(3):173-6.Ouyang P, Liu YL, Xu DL, et al. Effects of human wild-type p53,Rb, antisense human IGF-I and antisense rat ATIB gene transfer on neointimal proliferation after carotid artery injury in rat[J]. J First Mil Med Univ/Di Yi Jun Yi Da Xue Xue Bao, 2001, 21(3): 173-6. [9] Reis ED, Roque M, Dansky H, et al. Sulindac inhibits neointimal formation after arterial injury in wild-type and apolipoprotein E-deficient mice[J]. Proc Natl Acad Sci USA, 2000, 97(23): 12764-9. [10] Woodard GE, Rosado JA, Brown J. Expression and control of Ctype uatriuretic peptide in rat vascular smooth muscle cells[J]. Am J Physiol Regul Integr Comp Physiol, 2002, 282(1): R156-65. [11] Takeuchi H, Ohmori K, Kondo I, et al. Potentiation of C-type natriuretic peptide with ultrasound and microbubbles to prevent neointi mal formation after vascular injury in rats[J]. Cardiovasc Res, 2003,58(1): 231-8. [12] Mammo T, Nakaki T, Hishikawa K, et al. Natriuretic peptide-augmented induction of nitric oxide synthase through cyclic guanosine 3’,5’-monophosphate elevation in vascular smooth muscle cells[J].Endocrinology, 1995, 136(5): 2135-42. [13] Meurice T, Vallet B, Bauters C, et al. Role of endothelial cells in restenosis after coronary angioplasty[J]. Fundam Clin Pharmacol,1996, 10(3): 234-42. [14] Shinomiya M, Tashiro J, Saito Y, et al. C-type natriuretic peptide inhibits intimal thickening of rabbit carotid artery after balloon catheter injury[J]. Biochem Biophys Res Commun, 1994, 205(2):1051-6. [15] Hutchinson HG, Trindade PT, Cunanan DB, et al. Mechanisms of natriuretic-peptide-induced growth inhibition of vascular smooth muscle cells[J]. Cardiovasc Res, 1997, 35 (1): 158-67. [16] Doi K, Ikeda T, Itoh H, et al. C-Type natriuretic peptide induces redifferetiation of vascular smooth ceils with accelerated reendothelialization[J]. Arterioscler Thromb Vasc Biol, 2001, 21(6): 930-6. |